Zaporozhyan state medical university departement of infectious diseases



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47 

 

 



 

 

 



 

 

 



 

 

 



 

 

 



 

 

III. Epidemiological 



anamnesis 

 

 



 

 

 



 

 

 



3. Objectively 

examination 

 

 

 



 

 

I. General  



examination: 

- general state of the 

patient; 

-skin, mucous 

membranes; 

 

 



II. Gastrointestinal tract:  

- tongue; 

symptoms depend from the period of the 

disease, severity of the course, age of the 

patient, concomitant pathology 

 

 



Pay attention on: 

- temperature;  

- edema of the face; 

- itching; 

- skin rash; 

 

Pay attention on: 



- abdominal pain

- decreased of appetite, heartburn; 

- hepatomegaly; 

- meteorism; 

- jaundice; 

- symptoms of peritonitis; 

 

 

Pay attention on:  



- tachycardia; 

- decrease of arterial pressure; 

- muffed heart sound. 

- disorder of heart rhythm; 

 

 

- sleeplesness 



- irritation 

- fatique 

- dizziness 

- headache  

 



48 

- percussion and 

palpation of the 

abdomen; 

III. Cardiovascular 

system: 


IV. Nervous system 

3.  3. Prescribe 

laboratory 

and 


instrumental 

investigatio

ns, interpret 

the results 

of the 

investigatio



ns 

1. blood count. 

 

 

2. urinanalysis  



 

 

3.investigation of feci 



 

4. biochemical methods 

 

5. ultrasound 



investigation 

 

6. cholecystography  



  

7. fibro-esphago-gastro-

duodenoscopy 

Pay attention on anemia, leukocytosis, 

eosinophylia.  

 

  



 

  

Finding of the eggs or proglottids in feci 



 

Dysproteinemia, changes of the 

biochemical test (increase of total 

bilirubin and it’s fractures, ALT, AST 

and other) 

 

 



 

 

 



 

 

 

 

 

 

 

 

THE BEEF TAPEWORM 


49 

Etiology 

Taenairhynchus saginatus 



Epidemiology 

Anthroponosis. Peroral biohelmintiasis. 



 

The source of the invasion and definitive host is human. The intermediate 

host is cattle.  

 

The factor of the transmission is insufficient thermal  

processing of the meat 

Pathogenesis 

cysticercus  → small intestine → larvae fixate to mucous membrane  



 

 

 



In 2 month 2 месяца → adult worm → discharge 

of proglottids filled by eggs 



 

Mechanic action 

 

lesion of the mucous membrane in the site 



of the fixation of parazite 

 



irritation of the interoreceptors 

 



disorders of the absorbtion 

 



intoxication 

 



sensibilization 

 

 

 

 

Clinical  



manifestations 

  

 



abdominal pains (especially right 

hypogastrium area) 

 

nauses 



 

meteorism 



 

disorders of appetite 



 

disorders of stool 



 

hypoacidic gastritis 



 

it is typical the active exit of 



proglottids act of the defication 

(especially at the night) 

General toxic symptoms 

 



weakness 

 



fatique 

 



disorders of the sleep 

 



irritability 

 



headache  

 



dizziness 

 

Complications 

 

acute appendicitis acute  pancreatitis acute cholecystitis 



 

occlusive ileus 



Diagnostics 

General clinical methods 

 



moderately eosinophilia 

 



moderately anemia 

Specific methods 

 



revealation of the eggs and 

proglottids of helminth in the feci 



Treatment 

 



niclosamide paromomycin praziquantel   

 

Prophylaxis 

 

dehelminthization of the patients 



 

devastation of the soil 



 

veterinary-sanitary control 



 

the maintenance of the roles of the 



terminal processing of the meat  

PORK TAPEWORM AND CYSTICERCOSIS 


50 

Etiology 

Taenia solium   



Epidemiology 

Anthroponosis. 



 

Peroral biohelminthosis  



 

The source of the invasion and definitive host is human. The intermediate 

hosts are pigs, dogs, cats, sheep  

The factors of transmission is use into the food meat of the pig with cystecercs



Pathogenesis 

Taenia solium arised in use of the meat with larvae of the parasite. The adult 

helminth parasites in the organism of human. The human is the definitive 

host. Cysticercosis arises in the penetration of the eggs of helminth into the 

intestine. The human is an intermediate host 

 

Taenia soleum 

In infection by cystecercs in 2-3 

months  → adult helminth → 

small intestine  

Cystecercosis  

In infection by eggs of T. soleum in 2-2,5 

month  → cystecercs → brain, 

myocardium, skeletal musculature, eyes 

 

↓ 

↓ 



 

Mechanic influence  

 

 



the lesion of the mucous 

membrane in the site of the fixation of 

parasite 

 



irritation of interoreceptors  

 



disorder of the process of 

absorbtion 

 

intoxication 



 

sensibilization  



 

compression of the tissues 



 

inflammatory process around parasites  



 

toxic and allergic action   



 

Clinical 

manifestations 

  

 



moderate marked dyspeptic 

manifestations and general toxic 

syndrome 

 



the clinical manifestations is 

dependence on localization (brain, eyes, 

subcutaneous, cellulose)  

Diagnostics  

General clinic methods moderate eosinophilia 



Taenia soleum 

 



finding of the helminth’s eggs in the feci 

 



ovoscopy  

Cystecercosis  

In dependence on localization (ophtalmoscopy, roentgenogram, 

computed tomography) 

Serological methods 

ELISA 

Treatment 

Taenia soleum- Filicis maris acterium 

 Cystecercosis - surgical 

 



Prophylaxis Relevation and treatment of the patients: general sanitary measures; veterinary 

control 



 

DIPHILLOBOTHRIASIS 


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